Alzheimer’s Disease Research Paper Essay Example

Provided the gravity of this epidemic and its serious social and economic costs, public health should take more proactive action towards dealing with it. The plan should focus on prevention as well as treatment to ensure high effectiveness. It was updated in The plan contains two important aspects: research and care providing.

In my own practice, I will remain up to date and also work towards to achieving the goals of the plan set out by HHS. In addition, I will take the following steps with patients to whom I shall provide care. The social and economic impact of this disease make it an issue that should be a concern of many professionals beyond the health sphere. Collaboration from the scientific community is required, but collaborations from different agents from a wide spectrum of fields could lead to remarkable breakthroughs.

Making the best of our available resources is essential. And there is a lot of potential in a largescale collaboration to finally reach an Alzheimer-free sociology of alzheimer% 27s disease. Ali, N. Crews, L. Helmut M. Brought to you by SiteJabber. Search for:. Background The homeschool free worksheets is named after Dr Alois Alzheimer, who discovered it back inwhen one of his patients died of a mental illness that was unknown back then.

Signs: The signs of Alzheimer could include difficulty sleeping, significant mood changes, loss of memory, and increasing confusion. Surveillance methods The current surveillance methods of the disease include collecting and compiling data on the occurrence of the disease, related risk factors and health behaviors, in addition to burden of the disease. Blennow, O. Hansson and U.

What is Alzheimer's Disease? - CDC

Content type: Research. Published on: 13 February Biomarkers are increasingly employed for effective research into neurodegenerative diseases. They have become essential for reaching an accurate clinical diagnosis, monitoring disease, and refining entry crite Authors: Ayesha Khatun, Ross W. Content type: Review.

Published on: 4 February The development of novel digital tools and assessments is likely to increase the need for these Authors: Richard Milne and Carol Brayne.

Content type: Debate. Published on: 31 January Jin and Angelica Quartino. Published on: 22 January Authors: Adam P. Mecca, Julia W. McDonald, Hannah R. Michalak, Tyler A. Godek, Joanna E. Harris, Erika A.

Sociology of alzheimer s disease

Pugh, Emily C. Carson, Stephen M. Strittmatter and Christopher H. Published on: 18 January Published on: 14 January Published on: 13 January Its increased peptide aggregation propensity and toxicity make Le, Barbara J. Caldarone, Jeffrey L.

Published on: 10 January To date, no symptomatic treatment is available for patients with vascular cognitive impairment VCI. Authors: Jolien F. Klaassen, Anna E. Leeuwis, Philip Scheltens, Henry C.

Weinstein, Joop M. Published on: 7 January Mounting evidence links poor sleep quality with a higher risk of late-life dementia. However, the structural and cognitive correlates of insomnia are still not well understood.

The study aims were to character Fluctuations are one of the core clinical features characterizing dementia with Lewy bodies DLB. They represent a determining factor for its diagnosis and strongly impact the quality of life of patients and Given the rising prevalence and mortality of AD coupled with the growing total healthcare costs, there continues to be a sense of urgency in the medical community to develop effective means for the early diagnosis and successful treatment of this progressive neurodegenerative disease.

F Faculty Reviews are commissioned from members of the prestigious F Faculty and are edited as a service to readers. In order to make these reviews as comprehensive and accessible as possible, the referees provide input before publication and only the final, revised version is published.

The referees who approved the final version are listed with their names and affiliations but without their reports on earlier versions any comments will already have been addressed in the published version.

National Center for Biotechnology InformationU. Journal List FRes v. Version 1. Published online Jul Author information Article notes Copyright and License information Disclaimer. Competing interests: Jason Weller has no competing interests to declare. Andrew Budson is a consultant and speaker for General Electric, Lilly, and Axovant and is a clinical trial investigator for Biogen, Lilly, vTv therapeutics, and Axovant. Accepted Jul This is an open access article distributed under the terms of the Creative Commons Attribution Licence, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work resume scientist phd properly cited.

This article has been cited by other articles in PMC. Keywords: Alzheimer's disease, dementia, amyloid, tau. Figure 1. Open in a separate window. Current treatment At present, only two classes of pharmacologic therapy are available for patients with AD.

Anti-tau Since p-tau appears to be the downstream pathology and is likely the direct cause of symptoms in AD, drugs to reduce the burden of this protein are also in development Table 1. Summary As recently asthe diagnosis and management of AD relied upon clinical symptom reporting that fit the pattern of memory dysfunction and loss of functional independence in multiple cognitive domains.

Notes [version 1; referees: 2 approved]. Funding Statement The author s declared that no grants were involved in supporting this work. References 1. Alzheimers Res Ther.

Alzheimer's Association: Alzheimer's disease facts and figures. Alzheimers Dement. J Alzheimers Sociology of alzheimer s disease. Mossello E, Ballini E: Management of patients with Alzheimer's disease: pharmacological treatment and quality of life.

Ther Adv Chronic Dis. Potential subjects should be aware, however, that they may not receive their individual results of these experimental markers. To date, molecular profiling and imaging for markers associated with AD have not been shown to be sociology of alzheimer s disease with a clinical diagnosis of normal cognition, MCI, or AD.

Given the instability of the diagnosis of MCI, clinicians should closely follow identified patients with cognitive screens every months and be alert for signs of functional decline that indicate conversion to AD or other dementias. There are no FDA-approved therapies for MCI; therefore, the decision is to watch and wait or to consider off-label treatment with an acetylcholinesterase AChE inhibitor.

Mediterranean diet, physical exercise, and the maintenance of cardiovascular health have been associated in various epidemiologic studies with better cognitive outcomes. Randomized trials, however, have not been completed, and there are no data demonstrating the prevention of MCI or AD. Nevertheless, given the low risk of these interventions and the possible general health benefits, patients should be counseled on these measures.

Finally, as part of standard clinical care, advance directives and financial matters should also be discussed while patients are able to articulate their decisions, independent of their likelihood of conversion to dementia. Advance directives are discussed in more detail below. Research is now focused on improving the accuracy of identification of the subgroup of patients who have MCI and will progress to AD. Those patients who are interested in research or clinical-trial participation should be referred to a nearby ADRC or Web sites for clinical trial recruitment.

Currently, there are 5. As per the Diagnostic and Statistical Manual of Mental Disorders IV, Text Revision, AD requires the following: 1 new onset memory impairment; 2 another cognitive disturbance, such as aphasia, apraxia, agnosia, or executive functioning; and 3 a gradual, progressive course that results in significant functional impairment.

The criteria for possible AD apply if there is an atypical course or presentation, or there is another illness present but it is not the primary cause of the dementia. A definite diagnosis can only be made if there is a clinical presentation consistent with AD and there is neuropathological confirmation by autopsy or biopsy. The diagnosis of AD is made by clinical history, documentation of functional decline, and ruling out other potential etiologies of cognitive decline.

The differential diagnosis of AD includes other types of dementia mixed, vascular dementia, Lewy body dementia, frontotemporal dementia, normal pressure hydrocephalus, HIV, and other rare forms of dementiadelirium, and, less frequently, seizure disorders. Features that may indicate another type of dementia other than AD are tok essay help 2013 in Table 3. If the clinician suspects a dementia other than or in addition to an AD or a vascular etiology, a specialist referral should be considered for accurate diagnosis, as this significantly affects prognosis and treatment efficacy.

Of course, interest in participating in clinical trials would also trigger a referral. The patient declines an off-label trial of an AChE inhibitor, but does agree to exercise more and follow a Mediterranean diet. Her laboratory tests are unremarkable, as is a computed tomography scan of her brain. Two years later, she comes to her annual checkup accompanied by her daughter. The daughter is upset because on visiting her mother, she found multiple notices about unpaid utility bills, and the electric company has threatened to turn off the lights.

She is able to draw the clock correctly but is not able to fill in the time 10 minutes to You interview the daughter alone. The daughter describes piles of unopened bills from the past few months and is worried that her mother forgets to take her medications consistently. She notes that her mother repeats herself more frequently, and has had more difficulty with word-finding. The patient continues to attend to her ADLs without any difficulties. Should we be participating in research?

The putative mechanism is inhibition of the enzyme AChE, which transiently increases levels of acetylcholine at the synapse. The most commonly prescribed oral AChEI is donepezil. Bradycardia is the most concerning side effect, and an electrocardiogram should be done to rule out bradycardia or a bundle branch block. The most common side effects are GI upset and sleep disturbances such as vivid dreams, wakefulness, or sedation; these can be avoided or addressed by starting at a low dose and then slowly titrating up, taking medication with food, and switching the timing of the medication.

Patients who continue to experience GI side effects or who are not able to take oral medication can consider the rivastigmine transdermal patch Exelon. It is not curative, and patients continue to decline at the same rate as previously. Due to the progressive nature of AD and the profound effect on both the patients and families, it is important to provide education and either provide or refer for counseling.

Because most patients with early AD are able to participate in decision-making, this is a critical period to appoint a healthcare proxy and express their long-term healthcare goals. Patients should be encouraged to discuss wishes for their current situations but also to communicate at what point in their illness they would not want aggressive measures pursued any further.

Patients should also be encouraged to make research advance directives regarding what types of procedures or novel medications they would be willing to participate in. Goals of care and directives should be reviewed at least once annually with the family and, whenever possible, with the patient, or when there is a significant medical event particularly hospitalization complicated with delirium or loss of functioning.

Patients should also be counseled that they should decide whom to appoint as their power of attorney, and clinicians should remind families that power of attorney is a separate role from healthcare proxy. Future planning should include healthcare proxy, advance directives, and recommendation to obtain help with financial planning and power of attorney. All patients who drive and are newly diagnosed with dementia should be assessed for their driving safety. Clinical history and clinician judgment should be used to make a determination; MMSE scores have limited predictive value.

Some states have mandatory reporting of unsafe driving concerns. If the patient continues to drive, it is important to follow up with further questioning periodically. In the present case, the daughter should be educated that given the relatively late onset of dementia in her mother and her aunt, their age and medical conditions are more likely significant contributors to their dementia than their genetics.

Despite significant lay concern about the role of genes in AD, clinicians should remind family members that the most significant risk factor for all dementias is age. Families are often excited to participate in research, both for altruistic reasons as well as for the possibility of being exposed to a new agent sooner and in a safe manner. Clinical research has become an important focus for better understanding the course of MCI and AD and the development of future treatment and prevention.

Interested clinicians and caregivers are referred to the Web sites in Table 2. In addition, there are numerous longitudinal studies, usually offered through ADRCs, that offer autopsy diagnosis and brain-tissue donation for interested patients and families. You discuss your diagnosis of early stage AD with the patient and daughter, and they agree to start donepezil 5 mg once a day, which she tolerates well, and is able to increase to 10 mg in a month.

She gets out of bed late and spends hours staring at the TV instead of pursuing activities. When friends and family come to visit, she sits quietly and seems less interested in their jokes or stories. The daughter has taken over her finances. You ask the patient and the daughter to come to the office. The patient appears calm, although she describes herself as frustrated about her memory.

They both deny any changes in appetite or sleep. The physical examination is unremarkable. Everything is fine. Neuropsychiatric symptoms are commonly seen in MCI and AD and cluster into specific groups: apathetic syndrome, affective syndrome anxiety and depressionpsychomotor agitation, irritability, and aberrant motor behaviorpsychotic delusions and hallucinationsand manic disinhibition and euphoria syndromes.

Because there are no FDA-approved treatments for these syndromes, it is crucial for clinicians to monitor the efficacy of clinical interventions by documenting sociology of alzheimer% 27s disease detail the frequency and severity of symptoms. Depression and anxiety are common, especially during the initial stages when patients may have insight and are aware of their memory deficits.

Depression itself is a well-established risk factor for dementia; recurrent depressive symptoms appear to increase the risk for dementia but not MCI. The gold standard for the diagnosis of depression should include a carefully structured history from both the patient and informant.

Questions should focus on depressed mood, changes in sleep and appetite, hopelessness, worthlessness, and suicidality. The GDS is available in and item versions, and is also useful as a tool to monitor treatment effect. The other likely differential diagnosis is apathetic syndrome, which caregivers may perceive as depression.

However, unlike depression, the patient will not complain of sadness, and when strongly encouraged to engage in activities, will eventually participate, and may even enjoy them. Treatment of depression and apathy consists of nonpharmacologic and, when appropriate, pharmacologic approaches. Nonpharmacologic interventions for depression include family therapy, psychoeducation for the alzheimer s disease papers and family, and structured activities such as day programs and daily exercise regimens.

If the patient truly meets criteria for depression, the selection of an antidepressant is usually based on the side effect profile because the efficacy is similar among all agents. Although the common action among all the antidepressants is the increase of monoamine neurotransmitters, particularly serotonin and norepinephrine, this may not be the therapeutic mode of action, because an antidepressant effect is not observed until at least 1 and usually weeks later.

Common side effects of these classes include nausea, vomiting, weight gain or, more rarely, weight losssexual dysfunction, insomnia or hypersomnia, dizziness, urinary retention, and blurry vision. More concerning but rarer adverse effects include akathesia and prolongation of QTc interval. A black box warning has been issued for SSRIs in children and adolescents, cautioning against increased suicidal ideation.

In the geriatric population, however, a significant reduction in death from suicide was seen after the introduction of SSRIs. Commonly used antidepressants are summarized in Graphic organizer persuasive essay grade 2 5.

Older antidepressants include the tricyclic antidepressants and monoamine oxidase inhibitors. Tricyclic antidepressants are not usually recommended in the elderly because of their risk of cardiac arrhythmias and anticholinergic side effects.

Monoamine oxidase inhibitors are also not used as first line because of the danger of hypertensive crisis if dietary restrictions are not followed carefully. Referral to psychiatry should be considered in patients with psychotic symptoms, treatment-resistant symptoms, or a history of suicide attempts or other major psychiatric illness. Referral for admission should be made if the patient is acutely a danger to themselves or others. Three months later, the daughter and patient return for follow-up; the patient appears stable with no behavioral changes.

She has not become clinically depressed. She attends a senior center 2 days per week and has a home health aide 4 hours a day for 3 days a week, who takes her out on walks, to visit friends, and shopping. The daughter visits the patient on the weekends. Two years later, the daughter calls you because the patient has been accusing her of stealing money and sociology of alzheimer s disease aide of taking various personal belongings. Now I have to take care of mother when the home attendant is not there.

The physical examination is unremarkable; she shows no sign of new medical illness or infection.In the present enquiry we examined the effect of the CB agonist binds to receptor. Proceedings of the National Academy of Sciences Although cannabidiol was similar in neuroprotective capacity to BHT, cannabidiol has no known tumor-promoting effects. Furthermore, the ability of cannabidiol to protect against neuronal injury without inhibiting NMDAr may reduce the occurrence of toxicity or side effects associated with NMDAr antagonists blockers.

Previous studies have indicated that cannabidiol is not toxic, even when chronically administered to humans or given in large acute doses. Campbell and Gowran. British Journal of Pharmacology Current AD drugs, which serve as AChE neuro-transmitter inhibitors, have an assignment about alzheimer disease of unpleasant side effects such as hepatotoxicity and gastrointestinal disturbances. Manipulation of the cannabinoid pathway offers a new pharmacological approach for the treatment of AD that may be more efficacious than current treatment regimes.

Phyto-cannabinoids clearly offer a multifaceted approach for the essays on domestic violence causes of AD and future studies should focus on examining the efficacy of phyto-cannabinoids in the array of animal models that exhibit AD-like pathology and cognitive decline. In conclusion, manipulation of the cannabinoid system offers the potential to upregulate neuroprotective mechanisms while dampening neuroinflammation.

International Journal of Geriatric Psychiatry Today's treatment options are custom admission essay and limited due to substantial side effects. Motor activity was measured objectively using actigraphy. These findings were corroborated by improvements in Neuropsychiatric Inventory total score as well as in subscores for agitation, aberrant motor, and nighttime behaviors.

No side effects were observed. BBC News. August 21, the year of Researchers assessed their cognitive skills and looked at how they coped with daily life. They were also given anti-psychotic drugs, which reduce delusions and have a calming effect, and at least four other medications to control behaviour.

Free Essays words 2. Currently in the United States, there are 5. The main deficit that AD has on the brain is the deterioration of different areas of the brain Powerful Essays words 5 pages Preview. A possible condition for insomnia is Alzheimer disease AD. There is a risk of circadian rhythm disruption in people that may possibly have AD, because this leads to an increase in beta-amyloid plaques in the brain.

This is related to cognitive loss as AD progresses Kang et al, AD has no definitive test. Sociology of alzheimer% 27s disease and urine samples are tested so other medical cases are ruled out. If AD is suspected, the patient is made to undergo brain imaging, most likely a magnetic resonance imaging MRI Although there are registered treatments for AD, the number is still not enough.

In order to achieve this purpose, the understanding of pathology is imperative Better Essays words 2 pages Preview. The disease first appears around the age of sixty. Studies have concluded that as many as 5. This disease consists of having a difficult time focusing, doing daily activities, and feeling confused or lost Understanding Alzheimer 's Disease: the Basics, According to the scientist there are few causes of this disease. Alois Alzheimer made the decision to utilize what was a new staining technique on a sample from a deceased patient.

Upon staining, he noted what appeared to be abnormal for the current findings. The nerve cells were bunched up, having the appearance of knots. Within these bunches of nerve cells were also what is now known as plaques. Later, in a medical journal, Dr. This disease named after Dr.

Alois Alzheimer a German psychiatrist who discovered the pathological disorder of dementia. Definition of Alzheimer 's Disease Alzheimer 's is a progressive, degenerative disorder that attacks the brain 's neurons or nerve cells, resulting in loss of memory, thinking and behavioral changes and language skills There are primarily two types of nerve damage that occur in the brain; Amyloid plaques begin to build up in the brain as well as nerve cells begin to tangle up, causing neurofibrillary tangles.

Symptoms such as memory loss, behavioral changes, among others, and ultimately ending in death Younger individuals may have problems with memory, thinking and concentration Therefore, the brain, the hub of the central nervous system, is responsible for integrating all sensory and motor patterning. To understand the mechanisms of neurobiology it is often useful to observe the nervous system at the level of the neuron. Integration and communication between neurons is facilitated by neurotransmitters, chemicals which act as intermediaries at the synaptic gap Delcomyn, These results are sorted by most relevant first ranked search.

You may also sort these by color rating or essay length. Alzheimers Disease - Alzheimer's Disease Introduction to Alzheimer's Alzheimer's disease is a progressive degenerative disease of the brain. Alzheimers Disease - We are currently living in the age of technology. Alzheimers Disease alzheimer% 27s disease paper Memory loss, like old age is a condition which mankind has always reluctantly recognized and always - with resignation.

The Chronic and Incurale Disease of Alzheimers Alzheimers Disease Health Promotion Case Study - Introduction This section will discuss the impact of Alzheimer's disease on racial, cultural, and gender variables, with the focus being on the various approaches to care of the disease. Alzheimers Disease - Alzheimer 1 It is inevitable that eventually each of us will grow old and begin to face more and more health problems as our age rises.

Large Amounts of Glutamine as the Cause of Disease - Neurodegenerative disorders such as Alzheimer's disease and Parkinson's disease are caused by the aggregation of abnormal proteins in neurons.

Symptoms And Treatment Of Alzheimer 's Disease - As we age our bodies begin to break down, but occasionally an abnormal breakdown occurs. Alzheimer 's Disease : The Most Common Form Of Dementia - Alzheimer 's Disease Imagine this, you walk into your mother 's room one day the smell of candles fills the air, you can hear her humidifier going off, and she blankly stares at you as you walk in. Alzheimer's Disease: What are assignment about alzheimer disease Forgetting?

Alzheimer 's Disease : A Progressive Disease - Throughout the lifespan of a human being, your body will only continue to grow and deteriorate as time progresses. Alzheimer's Disease - We live in an advanced world of technology and medicine, with researcher constantly learning more about the diseases that lay waste to the world. Alzheimer's Disease - Even in a world of modern medicine and major medical advancements like the world has never seen before, some diseases still continue to plague the human race and confuse even some of the brightest scientists today.

Alzheimers The Unsolved Mystery - Alzheimer's: The Unsolved Mystery Absentmindedness, with questions having to be repeated, trouble following conversations, or remembering people's names, sound familiar. Pick's Disease - Pick's Disease Pick's disease is a form of dementia characterized by a progressive and irreversible deterioration of social skills and changes in personality, along with impairment of intellect, memory, and language.

Lowering The Rates Of Dementia - The rates of dementia differ greatly around the world, from the lowest rate in Africa and India, south Asia to the highest rate in western Europe, specialty north America, the incidence of dementia and Alzheimers disease significantly lower for African and Nigeria than Indian, about 5 times lower. The Disease Of Alzheimer 's Disease - Alzheimer disease, sources and warning sings, explanation of sickness as an intellect breakdown.

The Death Of Alzheimer 's Disease - The loss of a loved one is one of the most sociology of alzheimer% 27s disease events in life that everybody, inevitably, has to face. Searching for the Cure for Alzheimer's Disease - The human brain is a remarkable organ, complex chemical and electrical processes take place within our brains. Symptoms And Symptoms Of Alzheimer 's Disease - Dementia is the word that is used often times to describe conditions that cause memory loss.

Symptoms And Symptoms Of Alzheimer 's Disease - Dementia is a broad term used to describe conditions that cause memory loss. Alzheimer's Disease in the Elderly - Alzheimer is a disease that affects the elderly most.

Sociology of alzheimer% 27s disease

Diagnosis Of Alzheimer 's Disease - Have you ever forgotten something like where you put your keys or to pick up shampoo from the store. Alzheimer : A Disease Of Old Age - Introduction Alzheimer is a dementia that causes problems with behaviour, thinking and the memory in general. The, Alzheimer 's Disease Ad - Memories allow humans and animals to function by retrieving and retaining information that our senses gather throughout everyday life.

Alzheimer's Disease and Aluminium - Aluminium is present in the earth's crust in huge amount, mainly in the form of insoluble aluminium silicates. The Chemistry Of Alzheimer 's Disease - Alois Alzheimer, a German psychiatrist who discovered the disorder of dementia and diagnosed the condition.

How an outsider in Alzheimer’s research bucked the prevailing theory - and clawed for validation

Treatment for Alzheimer's Disease - Alzheimer's disease is an acquired impairment of cognitive and behavioral functioning of unknown cause that can evolve into dementia. Insomnia and Alzheimer Disease - 1. Alois Alzheimer 's Disease - Dr. Factors, Symptoms and Treatment of Alzheimer's Disease - Alzheimer Disease 1 Alzheimer Disease Introduction This research paper will examine factors, signs of symptoms, treatment, when to visit a doctor and how to care for a love one with Alzheimer disease.

Over the years, there has been widespread research that has been devoted towards explaining the configuration of SPs and NFTs to finds indications concerning the molecular pathogenesis and biochemistry of the disease. NFTs are mostly composed of the protein tau, which has a strong link to microtubules. The starch like substance is in turn surrounded by a thin layer of neuritis and reactive glia that disappear after some time.

In recent days, assignment about alzheimer disease have been able to characterize the protein as well as clone the gene encoding the precursor brain, thus bringing a lot of wealth in trying to demystify the disease. This test is ideal as it helps in identifying the areas whether the patient needs assistance in conducting these six basic activities. This test measures the ability of Alzheimer's disease and multi-infarct dementia can exist together, making it hard for the doctor to diagnose either one specifically.

Scientists use to think that multi-infarct dementia and other types of vascular dementia caused most cases of mental impairment. They now think that most older people with irreversible dementia have Alzheimer's disease. Wellness and Prevention There is no certain way to prevent Alzheimer's disease. It may be possible to reduce the risk of developing the illness, Because Alzheimer's disease occurs late in life and there is such a long interval between the start of symptoms and death, the ability to delay the time when the first problems become visible would be useful As people age, it is a good idea to do everything to protect their brains from premature failure.

This will allow them to function at. Read More. Words: - Pages: 5. Words: - Pages: 7. Alzheimer s disease papers - Pages: 4. Alzheimer's Disease and Aluminium Essay vulnerable to the development of Alzhiemer disease. Words: - Pages: 6. Alzheimer's Disease Essay Alzheimer's Disease Incomplete Works Cited Every era has its disease; the one debilitating killer that each individual grows up to fear and detest more than War or poverty or depression.

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